GBA Mutations in Parkinson Disease: Earlier Death But Similar Neuropathological Features
Department
neurology
Document Type
Article
Abstract
Background and purpose: Mutations in the glucocerebrosidase (GBA) gene are known to be a risk factor for Parkinson's disease (PD). Data on clinicopathological correlation are limited. The purpose of this study was to determine the clinicopathological findings that might distinguish PD cases with and without mutations in the GBA gene. Methods: Data from the Arizona Study of Aging and Neurodegenerative Disorders were used to identify autopsied PD cases that did or did not have a GBA gene mutation. Clinical and neuropathological data were compared. Results: Twelve PD cases had a GBA mutation and 102 did not. The GBA mutation cases died younger (76 vs. 81 years of age) but there was no difference in disease duration or clinical examination findings. No neuropathological differences were found in total or regional semi-quantitative scores for Lewy-type synucleinopathy, senile plaques, neurofibrillary tangles, white matter rarefaction or cerebral amyloid angiopathy scores. Conclusions: In longitudinally assessed, autopsied PD cases, those with GBA mutations had a younger age at death but there was no evidence for clinical or neuropathological differences compared to cases without GBA mutations. Due to the small GBA group size, small differences cannot be excluded.
Medical Subject Headings
neurology
Publication Date
2017
Publication Title
European Journal of Neurology
ISSN
1351-5101
Volume
24
Issue
11
First Page
1363
Last Page
1368
Digital Object Identifier (DOI)
10.1111/ene.13395
Recommended Citation
Adler, C. H.; Beach, T. G.; Shill, Holly A.; Caviness, John N.; Driver-Dunckley, E.; Sabbagh, M. N.; Patel, A.; Sue, L. I.; Serrano, G.; Jacobson, S. A.; Davis, K.; Belden, C. M.; Dugger, B. N.; Paciga, S. A.; Winslow, A. R.; Hirst, W. D.; and Hentz, J. G., "GBA Mutations in Parkinson Disease: Earlier Death But Similar Neuropathological Features" (2017). Neurology. 223.
https://scholar.barrowneuro.org/neurology/223