The septin CDCrel-1 binds syntaxin and inhibits exocytosis
Document Type
Article
Abstract
Septins are GTPases required for the completion of cytokinesis in diverse organisms, yet their roles in cytokinesis or other cellular processes remain unknown. Here we describe studies of a newly identified septin, CDCrel-1, which is predominantly expressed in the nervous system. This protein was associated with membrane fractions, and a significant fraction of the protein copurified and coprecipitated with synaptic vesicles. In detergent extracts, CDCrel-1 and another septin, Nedd5, immunoprecipitated with the SNARE protein syntaxin by directly binding to syntaxin via the SNARE interaction domain. Transfection of HIT-T15 cells with wild-type CDCrel-1 inhibited secretion, whereas GTPase dominant-negative mutants enhanced secretion. These data suggest that septins may regulate vesicle dynamics through interactions with syntaxin.
Publication Date
5-1-1999
Publication Title
Nature Neuroscience
ISSN
10976256
Volume
2
Issue
5
First Page
434
Last Page
439
PubMed ID
10321247
Digital Object Identifier (DOI)
10.1038/8100
Recommended Citation
Beites, Crestina L.; Xie, Hong; Bowser, Robert; and Trimble, William S., "The septin CDCrel-1 binds syntaxin and inhibits exocytosis" (1999). Translational Neuroscience. 618.
https://scholar.barrowneuro.org/neurobiology/618